PKD2L1/PKD1L3 channel complex with an alkali-activated mechanism and calcium-dependent inactivation-Reference-Cited by-同舟云学术

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PKD2L1/PKD1L3 channel complex with an alkali-activated mechanism and calcium-dependent inactivation

Published:2015-06-12 Issue:6 Volume:44 Page:483-492
ISSN:0175-7571
Container-title:European Biophysics Journal
language:en
Short-container-title:Eur Biophys J

Author:

Chen Peihua,Wu Jin-zhi,Zhao Jie,Wang Ping,Luo Jianhong,Yang Wei,Liu Xiao-dong

Funder

National Basic Research Program of China Grant

Natural Science Foundation of China grant

China National Key Basic Research Program grant

the National Basic Research Program of China Grant

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Biophysics

Link

http://link.springer.com/content/pdf/10.1007/s00249-015-1040-y.pdf

Reference39 articles.

1. Beg AA, Ernstrom GG, Nix P et al (2008) Protons act as a transmitter for muscle contraction in C. elegans. Cell 132(1):149–160

2. Budde T, Meuth S, Pape HC (2002) Calcium-dependent inactivation of neuronal calcium channels. Nat Rev Neurosci 3(11):873–883

3. Cai Y, Anyatonwu G, Okuhara D et al (2004) Calcium dependence of polycystin-2 channel activity is modulated by phosphorylation at Ser812. J Biol Chem 279(19):19987–19995

4. Caterina MJ, Schumacher MA, Tominaga M et al (1997) The capsaicin receptor: a heat-activated ion channel in the pain pathway. Nature 389(6653):816–824

5. Chandrashekar J, Kuhn C, Oka Y et al (2010) The cells and peripheral representation of sodium taste in mice. Nature 464(7286):297–301

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