Calcium pyrophosphate dihydrate and hydroxyapatite crystal deposition in the joint: New developments relevant to the clinician
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://link.springer.com/content/pdf/10.1007/s11926-003-0073-x.pdf
Reference92 articles.
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2. Jaovisidha K, Rosenthal AK: Calcium crystals in osteoarthritis. Curr Opin Rheumatol 2002, 14:298–302.
3. Johnson K, Hashimoto S, Lotz M, et al.: Up-regulated expression of the phosphodiesterase nucleotide pyrophosphatase family member PC-1 is a marker and pathogenic factor for knee meniscal cartilage matrix calcification. Arthritis Rheum 2001, 44:1071–1081. Using immunohistochemistry and transfection studies with the three different NTPPPH isoenzymes, the authors identify PC-1 as a direct pathogenic factor for cartilage matrix calcification.
4. Johnson K, Vaingankar S, Chen Y, et al.: Differential mechanisms of inorganic pyrophosphate production by plasma cell membrane glycoprotein-1 and B10 in chondrocytes. Arthritis Rheum 1999, 42:1986–1997. The authors demonstrated the mechanism for TGFβ-induced extracellular PPi elevation to involve PC-1 expression and translocation to the plasma membrane.
5. Terkeltaub R: Inorganic pyrophosphate generation and disposition in pathophysiology. AJP Cell Physiol 2001, 281:C1-C11. This comprehensive review summarizes recent developments in the understanding of the generation and disposal of PPi and discusses the role of PPi metabolism in diseases with connective tissue calcification.
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