Metabolic Regulation of Tendon Inflammation and Healing Following Injury
Author:
Funder
National Institute of Arthritis and Musculoskeletal and Skin Diseases
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://link.springer.com/content/pdf/10.1007/s11926-021-00981-4.pdf
Reference94 articles.
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2. Birch HL, Rutter GA, Goodship AE. Oxidative energy metabolism in equine tendon cells. Res Vet Sci. 1997;62(2):93–7.
3. Nichols AEC, Settlage RE, Werre SR, Dahlgren LA. Novel roles for scleraxis in regulating adult tenocyte function. BMC Cell Biol. 2018;19(1):14.
4. Izumi S, Otsuru S, Adachi N, Akabudike N, Enomoto-Iwamoto M. Control of glucose metabolism is important in tenogenic differentiation of progenitors derived from human injured tendons. PLoS One. 2019;14(3):e0213912 This study examines how metabolic flux through glycolysis and the TCA cycle differently influence differentiation potential of cells derived from human injured tendons.
5. Zhang K, Asai S, Yu B, Enomoto-Iwamoto M. IL-1β irreversibly inhibits tenogenic differentiation and alters metabolism in injured tendon-derived progenitor cells in vitro. Biochem Biophys Res Commun. 2015;463(4):667–72 This study demonstrated that pro-inflammatory cytokine IL-1β enhanced lactate production, directly affecting expression of tendon marker Scx and matrix gene Col1.
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