Dopamine depletion augments endogenous opioid-induced locomotion in the nucleus accumbens using bothμ1 andδ opioid receptors-Reference-Cited by-同舟云学术

Dopamine depletion augments endogenous opioid-induced locomotion in the nucleus accumbens using bothμ1 andδ opioid receptors

Published:1995-08 Issue:3 Volume:120 Page:347-355
ISSN:0033-3158
Container-title:Psychopharmacology
language:en
Short-container-title:Psychopharmacology

Author:

Churchill L.,Roques B. P.,Kalivas P. W.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology

Link

http://link.springer.com/content/pdf/10.1007/BF02311183.pdf

Reference47 articles.

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2. Bouboutou R, Waksman G, Devin J, Foiurnie-Zaluski M-C, Roques BP (1984) Bidentate peptides: highly potent new inhibitors of enkephalin degrading enzymes. Life Sci 35:1023–1030

3. Childers SR (1988) Opiate-inhibited adenylate cyclase in rat brain membranes depleted of Gs-stimulated adenylate cyclase. J Neurochem 50:543–553

4. Churchill L, Kalivas PW (1992) Dopamine depletion produces augmented behavioral responses to a mu, but not a delta-opioid agonist in the nucleus accumbens: lack of a role for receptor upregulation. Synapse 11:47–57

5. Churchill L, Austin MC, Kalivas PW (1992) Dopamine and endogenous opioid regulation of picrotoxin-induced locomotion in the ventral pallidum after dopamine depletion in the nucleus accumbens. Psychopharmacology 108:141–146

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