Changes in the Expression of Bcl-2 Protein in Neurons in the Hippocampal Fields after Use of Ischemic Postconditioning of the Brain

Author:

Shcherbak N. S.,Rusakova A. G.,Galagudza M. M.,Yukina G. Yu.,Barantsevich E. R.,Tomson V. V.,Shlyakhto E. V.

Publisher

Springer Science and Business Media LLC

Subject

General Neuroscience

Reference20 articles.

1. O. V. Vetrovoi, E. A. Rybnikova, T. S. Glushchenko, and M. O. Samoilov, “Effects of hypoxic postconditioning on expression of the antiapoptotic protein Bcl-1 and the neurotrophin BDNF in hippocampal field CA1 in rats subjected to severe hypoxia,” Morfologiya, 145, No. 2, 16–20 (2014).

2. N. S. Shcherbak, M. M. Galagudza, A. N. Kuz’menkov, et al., “Morpho functional changes in hippocampal field CA1 in Mongolian gerbils on use of ischemic postconditioning,” Morfologiya, 142, No. 5, 12–16 (2012).

3. Y. J. Cao, T. Shibata, and N. G. Rainov, “Liposome-mediated transfer of the bcl-2 gene results in neuroprotection after in vivo transient focal cerebral ischemia in an animal model,” Gene Ther., 9, No. 6, 415–419 (2002).

4. E. Castren, Y. Ohga, M. P. Berzaghi, et al., “bcl-2 messenger RNA is localized in neurons of the developing and adult rat brain,” Neuroscience, 61, 165–177 (1994).

5. Z. M. Ding, B. Wu, W. Q. Zhang, et al., “Neuroprotective effects of ischemic preconditioning and postconditioning on global brain ischemia in rats through the same effect on inhibition of apoptosis,” Int. J. Mol. Sci., 13, No. 5, 6089–6101 (2012).

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