ZINC40099027 promotes monolayer circular defect closure by a novel pathway involving cytosolic activation of focal adhesion kinase and downstream paxillin and ERK1/2-Reference-Cited by-同舟云学术

ZINC40099027 promotes monolayer circular defect closure by a novel pathway involving cytosolic activation of focal adhesion kinase and downstream paxillin and ERK1/2

Published:2022-08-24 Issue:2 Volume:390 Page:261-279
ISSN:0302-766X
Container-title:Cell and Tissue Research
language:en
Short-container-title:Cell Tissue Res

Author:

Oncel Sema^ORCID,Basson Marc D.^ORCID

Funder

National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Histology,Pathology and Forensic Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00441-022-03674-1.pdf

Reference84 articles.

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3. Balabanova L, Golotin V, Podvolotskaya A, Rasskazov V (2015) Genetically modified proteins: functional improvement and chimeragenesis. Bioengineered 6:262. https://doi.org/10.1080/21655979.2015.1075674

4. Basson MD, Coppola CP (2002) Repetitive deformation and pressure activate small bowel and colonic mucosal tyrosine kinase activity in vivo. Metabolism 51:1525–1527. https://doi.org/10.1053/meta.2002.36303

5. Basson MD, Modlin IM, Madri JA (1992) Human enterocyte (Caco-2) migration is modulated in vitro by extracellular matrix composition and epidermal growth factor. J Clin Invest 90:15–23. https://doi.org/10.1172/JCI115828

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