Idiopathic nephrotic syndrome and serum permeability factors: a molecular jigsaw puzzle
Author:
Funder
Fondation pour la Recherche Médicale
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Histology,Pathology and Forensic Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00441-019-03147-y.pdf
Reference120 articles.
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2. Alachkar N, Li J, Matar D et al (2018) Monitoring suPAR levels in post-kidney transplant focal segmental glomerulosclerosis treated with therapeutic plasma exchange and rituximab. BMC Nephrol:19. https://doi.org/10.1186/s12882-018-1177-x
3. Artero ML, Sharma R, Savin VJ, Vincenti F (1994) Plasmapheresis reduces proteinuria and serum capacity to injure glomeruli in patients with recurrent focal glomerulosclerosis. Am J Kidney Dis Off J Natl Kidney Found 23:574–581
4. Ashworth CT, James JA (1961) Glomerular excretion of macromolecular substances. Electron microscopic study of rat kidney after administration of human serum albumin. Am J Pathol 39:307–316
5. Audard V, Zhang S, Copie-Bergman C et al (2010) Occurrence of minimal change nephrotic syndrome in classical Hodgkin lymphoma is closely related to the induction of c-mip in Hodgkin-reed Sternberg cells and podocytes. Blood 115:3756–3762. https://doi.org/10.1182/blood-2009-11-251132
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