Vav Proteins in Cancer
Author:
Billadeau Daniel D.
Publisher
Springer New York
Reference69 articles.
1. Aghazadeh, B., Lowry, W. E., Huang, X. Y., and Rosen, M. K. (2000). Structural basis for relief of autoinhibition of the Dbl homology domain of proto-oncogene Vav by tyrosine phosphorylation. Cell 102, 625–633. 2. Banach-Petrosky, W., Jessen, W. J., Ouyang, X., Gao, H., Rao, J., Quinn, J., Aronow, B. J., and Abate-Shen, C. (2007). Prolonged exposure to reduced levels of androgen accelerates prostate cancer progression in Nkx3.1; Pten mutant mice. Cancer research 67, 9089–9096. 3. Bartolome, R. A., Molina-Ortiz, I., Samaniego, R., Sanchez-Mateos, P., Bustelo, X. R., and Teixido, J. (2006). Activation of Vav/Rho GTPase signaling by CXCL12 controls membrane-type matrix metalloproteinase-dependent melanoma cell invasion. Cancer research 66, 248–258. 4. Betz, R., Sandhoff, K., Fischer, K. D., and van Echten-Deckert, G. (2003). Detection and identification of Vav1 protein in primary cultured murine cerebellar neurons and in neuroblastoma cells (SH-SY5Y and Neuro-2a). Neuroscience letters 339, 37–40. 5. Booden, M. A., Campbell, S. L., and Der, C. J. (2002). Critical but distinct roles for the pleckstrin homology and cysteine-rich domains as positive modulators of Vav2 signaling and transformation. Molecular and cellular biology 22, 2487–2497.
|
|