Cross talk between 26S proteasome and mitochondria in human mesenchymal stem cells’ ability to survive under hypoxia stress-Reference-Cited by-同舟云学术

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Cross talk between 26S proteasome and mitochondria in human mesenchymal stem cells’ ability to survive under hypoxia stress

Published:2019-11 Issue:6 Volume:69 Page:1005-1017
ISSN:1880-6546
Container-title:The Journal of Physiological Sciences
language:en
Short-container-title:J Physiol Sci

Author:

Khasawneh Ramada R.,Abu-El-Rub Ejlal,Serhan Abdullah Omar,Serhan Bashar Omar,Abu-El-Rub Hadeel

Funder

Deanship of Research at Yarmouk University

Publisher

Springer Science and Business Media LLC

Subject

Physiology

Link

http://link.springer.com/content/pdf/10.1007/s12576-019-00720-6.pdf

Reference52 articles.

1. Abu-El-Rub E, Sequiera GL, Sareen N et al (2019) Hypoxia-induced 26S proteasome dysfunction increases immunogenicity of mesenchymal stem cells. Cell Death Dis 10:90. https://doi.org/10.1038/s41419-019-1359-x

2. Azad P, Haddad G (2017) Molecular basis of hypoxia induced excessive erythrocytosis. FASEB J 31:709.5. https://doi.org/10.1096/fasebj.31.1_supplement.709.5

3. Bard JAM, Goodall EA, Greene ER et al (2018) Structure and function of the 26S proteasome. Annu Rev Biochem 87:697–724. https://doi.org/10.1146/annurev-biochem-062917-011931

4. Box AH, Kim S-M, Demetrick DJ (2010) AKT loss in human epithelial cells treated with severe hypoxia. Biochim Biophys Acta 1803:951–959. https://doi.org/10.1016/j.bbamcr.2010.03.011

5. Capasso S, Alessio N, Squillaro T et al (2015) Changes in autophagy, proteasome activity and metabolism to determine a specific signature for acute and chronic senescent mesenchymal stromal cells. Oncotarget 6:39457–39468. https://doi.org/10.18632/oncotarget.6277

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