Oxygen–Glucose Deprivation Decreases the Motility and Length of Axonal Mitochondria in Cultured Dorsal Root Ganglion Cells of Rats
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Cellular and Molecular Neuroscience,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10571-022-01247-y.pdf
Reference54 articles.
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2. Baltan S (2014) Excitotoxicity and mitochondrial dysfunction underlie age-dependent ischemic white matter injury. Adv Neurobiol 11:151–170. https://doi.org/10.1007/978-3-319-08894-5_8
3. Bastian C, Politano S, Day J, McCray A, Brunet S, Baltan S (2018a) Mitochondrial dynamics and preconditioning in white matter. Cond Med 1:64–72
4. Bastian C, Zaleski J, Stahon K, Parr B, McCray A, Day J, Brunet S, Baltan S (2018b) NOS3 inhibition confers post-ischemic protection to young and aging white matter integrity by conserving mitochondrial dynamics and Miro-2 levels. J Neurosci 38:6247–6266. https://doi.org/10.1523/JNEUROSCI.3017-17.2018
5. Berbusse GW, Woods LC, Vohra BP, Naylor K (2016) Mitochondrial dynamics decrease prior to axon degeneration induced by vincristine and are partially rescued by overexpressed cytNmnat1. Front Cell Neurosci 10:179. https://doi.org/10.3389/fncel.2016.00179
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1. Correction to: Oxygen–Glucose Deprivation Decreases the Motility and Length of Axonal Mitochondria in Cultured Dorsal Root Ganglion Cells of Rats;Cellular and Molecular Neurobiology;2022-07-14
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