Mimicking Parkinson’s Disease in a Dish: Merits and Pitfalls of the Most Commonly used Dopaminergic In Vitro Models-Reference-Cited by-同舟云学术

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Mimicking Parkinson’s Disease in a Dish: Merits and Pitfalls of the Most Commonly used Dopaminergic In Vitro Models

Published:2017-07-18 Issue:2-3 Volume:19 Page:241-255
ISSN:1535-1084
Container-title:NeuroMolecular Medicine
language:en
Short-container-title:Neuromol Med

Author:

Lopes Fernanda Martins,Bristot Ivi Juliana,da Motta Leonardo Lisbôa,Parsons Richard B.,Klamt Fabio

Funder

CNPq/MS/SCTIE/DECIT - Pesquisas Sobre Doenças Neurodegenerativas

MCT/CNPq INCT-TM

Rapid Response Innovation Award/MJFF

MCT/CNPq

Programa de Doutorado Sanduíche no Exterior - PDSE/CAPES

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Molecular Medicine

Link

http://link.springer.com/article/10.1007/s12017-017-8454-x/fulltext.html

Reference145 articles.

1. Abad, F., Maroto, R., López, M. G., et al. (1995). Pharmacological protection against the cytotoxicity induced by 6-hydroxydopamine and H2O2 in chromaffin cells. European Journal of Pharmacology, 293, 55–64.

2. Agholme, L., Lindström, T., Kågedal, K., et al. (2010). An in vitro model for neuroscience: differentiation of SH-SY5Y cells into cells with morphological and biochemical characteristics of mature neurons. Journal of Alzheimer’s Disease, 20, 1069–1082. doi: 10.3233/JAD-2010-091363 .

3. Bal-Price, A. K., Hogberg, H. T., Buzanska, L., & Coecke, S. (2010). Relevance of in vitro neurotoxicity testing for regulatory requirements: Challenges to be considered. Neurotoxicology and Teratology, 32, 36–41. doi: 10.1016/j.ntt.2008.12.003 .

4. Bayir, H., Kapralov, A. A., Jiang, J., et al. (2009). Peroxidase mechanism of lipid-dependent cross-linking of synuclein with cytochrome c: Protection against apoptosis versus delayed oxidative stress in parkinson disease. Journal of Biological Chemistry, 284, 15951–15969. doi: 10.1074/jbc.M900418200 .

5. Beal, M. F. (2010). Parkinson’s disease: A model dilemma. Nature, 466, S8–S10. doi: 10.1038/466S8a .

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