Methylglyoxal Induces Tau Hyperphosphorylation via Promoting AGEs Formation-Reference-Cited by-同舟云学术

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Methylglyoxal Induces Tau Hyperphosphorylation via Promoting AGEs Formation

Published:2012-07-14 Issue:4 Volume:14 Page:338-348
ISSN:1535-1084
Container-title:NeuroMolecular Medicine
language:en
Short-container-title:Neuromol Med

Author:

Li Xiao-Hong,Xie Jia-Zhao,Jiang Xia,Lv Bing-Ling,Cheng Xiang-Shu,Du Lai-Ling,Zhang Jia-Yu,Wang Jian-Zhi,Zhou Xin-Wen

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s12017-012-8191-0.pdf

Reference49 articles.

1. Ahmed, M. U., Brinkmann, Frye. E., Degenhardt, T. P., Thorpe, S. R., & Baynes, J. W. (1997). N-epsilon-(carboxyethyl)lysine, a product of the chemical modification of proteins by methylglyoxal, increases with age in human lens proteins. The Biochemical Journal, 324, 565–570.

2. Ahmed, N., & Thornalley, P. J. (2003). Quantitative screening of protein biomarkers of early glycation, advanced glycation, oxidation and nitrosation in cellular and extracellular proteins by tandem mass spectrometry multiple reaction monitoring. Biochemical Society Transactions, 31, 1417–1422.

3. Alafuzoff, I., Iqbal, K., Friden, H., Adolfsson, R., & Winblad, B. (1987). Histopathological criteria for progressive dementia disorders: Clinical-pathological correlation and classification by multivariate data analysis. Acta Neuropathologica, 74, 209–225.

4. Brownlee, M., Vlassara, H., Kooney, A., Ulrich, P., & Cerami, A. (1986). Aminoguanidine prevents diabetes-induced arterial wall protein cross-linking. Science, 232, 1629–1632.

5. Butterfield, D. A. (2002). Amyloid beta-peptide (1–42)-induced oxidative stress and neurotoxicity: Implications for neurodegeneration in Alzheimer’s disease brain. A review. Free Radical Research, 36, 1307–1313.

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