Acetyl-11-keto-β-boswellic acid (AKBA) Attenuates Oxidative Stress, Inflammation, Complement Activation and Cell Death in Brain Endothelial Cells Following OGD/Reperfusion
Author:
Funder
Barrow Neurological Foundation
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology,Molecular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s12017-019-08569-z.pdf
Reference40 articles.
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3. Alluri, H., Anasooya Shaji, C., Davis, M. L., & Tharakan, B. (2015). Oxygen-glucose deprivation and reoxygenation as an in vitro ischemia-reperfusion injury model for studying blood–brain barrier dysfunction. Journal of Visualized Experiments. https://doi.org/10.3791/52699 .
4. Alluri, H., Stagg, H. W., Wilson, R. L., Clayton, R. P., Sawant, D. A., Koneru, M., et al. (2014). Reactive oxygen species-caspase-3 relationship in mediating blood–brain barrier endothelial cell hyperpermeability following oxygen-glucose deprivation and reoxygenation. Microcirculation,21(2), 187–195. https://doi.org/10.1111/micc.12110 .
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