Amplification of “defective” retrotransposon gtwin in D. melanogaster strain carrying large complex chromosomal aberration-Reference-Cited by-同舟云学术

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Amplification of “defective” retrotransposon gtwin in D. melanogaster strain carrying large complex chromosomal aberration

Published:2010-09-22 Issue:5 Volume:284 Page:373-381
ISSN:1617-4615
Container-title:Molecular Genetics and Genomics
language:en
Short-container-title:Mol Genet Genomics

Author:

Stefanov Yury E.,Glukhov Ivan A.,Kotnova Alina P.,Salenko Veniamin B.,Pasyukova Elena G.,Lyubomirskaya Natalia V.,Ilyin Yuriy V.

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00438-010-0573-0.pdf

Reference21 articles.

1. Aravin AA, Hannon GJ, Brennecke J (2007) The Piwi-piRNA pathway provides an adaptive defense in the transposon arms race. Science 318(5851):761–764

2. Bergman CM, Quesneville H, Anxolabéhère D, Ashburner M (2006) Recurrent insertion and duplication generate networks of transposable element sequences in the Drosophila melanogaster genome. Genome Biol 7(11):R112

3. Bingham PM, Kidwell MG, Rubin GM (1982) The molecular basis of P-M hybrid dysgenesis: the role of P-element, a P specific transposable family. Cell 29:995–1004

4. Bowen NJ, McDonald JF (2001) Drosophila euchromatic LTR retrotransposons are much younger than the host species in which they reside. Genome Res 11(9):1527–1540

5. Brennecke J, Aravin AA, Stark A, Dus M, Kellis M, Sachidanandam R, Hannon GJ (2007) Discrete small RNA-generating loci as master regulators of transposon activity in Drosophila. Cell 128(6):1089–1103

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