Psychobiological Pathways of PCOS
Author:
Publisher
Springer International Publishing
Link
http://link.springer.com/content/pdf/10.1007/978-3-030-30290-0_7
Reference80 articles.
1. Abbott, D. H., Dumesic, D. A., Eisner, J. R., Colman, R. J., & Kemnitz, J. W. (1998). Insights into the development of polycystic ovary syndrome (PCOS) from studies of prenatally androgenized female rhesus monkeys. Trends in Endocrinology & Metabolism, 9, 62–67.
2. Abbott, D. H., Dumesic, D. A., & Franks, S. (2002). Developmental origin of polycystic ovary syndrome—A hypothesis. The Journal of Endocrinology, 174, 1–5.
3. Abbott, D. H., Dumesic, D. A., & Levine, J. E. (2019). Hyperandrogenic origins of polycystic ovary syndrome–implications for pathophysiology and therapy. Expert Review of Endocrinology and Metabolism, 14(2), 131–143.
4. Altuntas, Y., Bilir, M., Ucak, S., & Gundogdu, S. (2005). Reactive hypoglycemia in lean young women with PCOS and correlations with insulin sensitivity and with beta cell function. European Journal of Obstetrics & Gynecology and Reproductive Biology, 119(2), 198–205.
5. Anderson, H., Fogel, N., Grebe, S. K., Singh, R. J., Taylor, R. L., & Dunaif, A. (2010). Infants of women with polycystic ovary syndrome have lower cord blood androstenedione and estradiol levels. The Journal of Clinical Endocrinology & Metabolism, 95(5), 2180–2186.
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