Connexins Biology in the Pathophysiology of Retinal Diseases
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Publisher
Springer International Publishing
Link
https://link.springer.com/content/pdf/10.1007/978-3-031-27681-1_33
Reference30 articles.
1. Söhl G, Willecke K. Gap junctions and the connexin protein family. Cardiovasc Res. 2004;62:228–32. https://doi.org/10.1016/j.cardiores.2003.11.013.
2. Ivanova E, Yee CW, Baldoni R Jr, Sagdullaev BT. Aberrant activity in retinal degeneration impairs central visual processing and relies on Cx36-containing gap junctions. Exp Eye Res. 2016;150:81–9. https://doi.org/10.1016/j.exer.2015.05.013.
3. Roy K, Kumar S, Bloomfield SA. Gap junctional coupling between retinal amacrine and ganglion cells underlies coherent activity integral to global object perception. Proc Natl Acad Sci. 2017;114:E10484–93. https://doi.org/10.1073/pnas.1708261114.
4. Banerjee S, Wang Q, Zhao F, Tang G, So C, Tse D, To, C.-H, Feng Y, Zhou X, Pan F. Increased Connexin36 phosphorylation in AII Amacrine cell coupling of the mouse myopic retina. Front Cell Neurosci. 2020;14 https://doi.org/10.3389/fncel.2020.00124.
5. Güldenagel M, Söhl G, Plum A, Traub O, Teubner B, Weiler R, Willecke K. Expression patterns of connexin genes in mouse retina. J Comp Neurol. 2000;425:193–201.
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