Pancreatitis, Rare Forms
Author:
Publisher
Springer International Publishing
Link
https://link.springer.com/content/pdf/10.1007/978-3-030-62416-3_5518
Reference11 articles.
1. Abraham, S. C., Leach, S., Yeo, C. J., Cameron, J. L., Murakata, L. A., Boitnott, J. K., Albores-Saavedra, J., & Hruban, R. H. (2003). Eosinophilic pancreatitis and increased eosinophils in the pancreas. American Journal of Surgical Pathology, 27(3), 334–342.
2. Carr, R. A., Rejowski, B. J., Cote, G. A., Pitt, H. A., & Zyromski, N. J. (2016). Systematic review of hypertriglyceridemia-induced acute pancreatitis: A more virulent etiology? Pancreatology, 16(4), 469–476. https://doi.org/10.1016/j.pan.2016.02.011.
3. Del Vecchio Blanco, G., Gesuale, C., Varanese, M., Monteleone, G., & Paoluzi, O. A. (2019). Idiopathic acute pancreatitis: A review on etiology and diagnostic work-up. Clinical Journal of Gastroenterology, 12(6), 511–524. https://doi.org/10.1007/s12328-019-00987-7.
4. Esposito, I., Hruban, R. H., Verbeke, C., et al. (2020). Guidelines on the histopathology of chronic pancreatitis. Recommendations from the working group for the international consensus guidelines for chronic pancreatitis in collaboration with the International Association of Pancreatology, the American Pancreatic Association, the Japan Pancreas Society, and the European Pancreatic Club. Pancreatology, 20(4), 586–593.
5. Frick, T. W. (2012). The role of calcium in acute pancreatitis. Surgery, 152(3 Suppl 1), S157–S163. https://doi.org/10.1016/j.surg.2012.05.013.
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