Interplay between LPL and GPIHBP1 in COVID-19 patients: a possible mechanism for post-recovery cardiomyopathy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology
Link
https://link.springer.com/content/pdf/10.1007/s13577-023-00953-z.pdf
Reference9 articles.
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2. Castiello T, Georgiopoulos G, Finocchiaro G, Claudia M, Gianatti A, Delialis D, Aimo A, Prasad S. COVID-19 and myocarditis: a systematic review and overview of current challenges. Heart Fail Rev. 2022;27:251–61. https://doi.org/10.1007/s10741-021-10087-9.
3. Abumrad NA, Cabodevilla AG, Samovski D, Pietka T, Basu D, Goldberg IJ. Endothelial cell receptors in tissue lipid uptake and metabolism. Circ Res. 2021;128:433–50. https://doi.org/10.1161/circresaha.120.318003.
4. Yagyu H, Chen G, Yokoyama M, Hirata K, Augustus A, Kako Y, Seo T, Hu Y, Lutz EP, Merkel M, et al. Lipoprotein lipase (LpL) on the surface of cardiomyocytes increases lipid uptake and produces a cardiomyopathy. J Clin Investig. 2003;111:419–26. https://doi.org/10.1172/jci16751.
5. Saxena U, Witte LD, Goldberg IJ. Release of endothelial cell lipoprotein lipase by plasma lipoproteins and free fatty acids. J Biol Chem. 1989;264:4349–55.
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