Intestinal barrier function is modulated by short-term exposure to fumonisin B1 in Ussing chambers
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Veterinary,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11259-009-9310-8.pdf
Reference13 articles.
1. Bock, J., Liebisch, G., Schweimer, J., Schmitz, G. and Rogler, G., 2007. Exogenous sphingomyelinase causes impaired intestinal epithelial barrier function. World journal of gastroenterology, 13, 5217–5225
2. Boudry, G., Lallès, J.P., Malbert, C.H., Bobillier, E. and Sève, B., 2002. Diet-related adaptation of the small intestine at weaning in pigs is functional rather than structural. Journal of pediatric gastroenterology & nutrition, 34, 180–187
3. Bouhet, S. and Oswald, I.P., 2007. The intestine as a possible target for fumonisin toxicity. Molecular nutrition and food research, 51, 925–931
4. Bouhet, S., Hourcade, E., Loiseau, N., Fikry, A., Martinez, S., Roselli, M., Galtier, P., Mengheri, E. and Oswald, I.P., 2004. The mycotoxin fumonisin B1 alters the proliferation and the barrier function of porcine intestinal epithelial cells. Toxicology science, 77, 165–171
5. Clarke, L.L., 2009. A guide to Ussing chamber studies of mouse intestine. American journal of physiology, 296, G1151–G1166
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