The glucocorticoid receptor gene polymorphism N363S predisposes to more severe toxic side effects during pediatric acute lymphoblastic leukemia (ALL) therapy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Hematology
Link
http://link.springer.com/content/pdf/10.1007/s12185-012-1236-1.pdf
Reference53 articles.
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2. Lee-Sherick AB, Linger RM, Gore L, et al. Targeting paediatric acute lymphoblastic leukaemia: novel therapies currently in development. Br J Haematol. 2010;151:295–311.
3. Mussai F, Campana D, Bhojwani D, et al. Cytotoxicity of the anti-CD22 immunotoxin HA22 (CAT-8015) against paediatric acute lymphoblastic leukaemia. Br J Haematol. 2010;150:352–8.
4. Stanulla M, Schrauder A. Bridging the gap between the north and south of the world: the case of treatment response in childhood acute lymphoblastic leukemia. Haematol Hematol J. 2009;94:748–52.
5. Tissing WJE, Meijerink JPP, den Boer ML, et al. Genetic variations in the glucocorticoid receptor gene are not related to glucocorticoid resistance in childhood acute lymphoblastic leukemia. Clin Cancer Res. 2005;11:6050–6.
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