GPS2 promotes erythroid differentiation in K562 erythroleukemia cells primarily via NCOR1
Author:
Funder
National Natural Science Foundation of China
National Key Research and Development Program of China
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s12185-024-03797-x.pdf
Reference34 articles.
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2. Zhang J, Kalkum M, Chait BT, Roeder RG. The N-CoR-HDAC3 nuclear receptor corepressor complex inhibits the JNK pathway through the integral subunit GPS2. Mol Cell. 2002;9(3):611–23. https://doi.org/10.1016/s1097-2765(02)00468-9.
3. Treuter E, Fan R, Huang Z, Jakobsson T, Venteclef N. Transcriptional repression in macrophages-basic mechanisms and alterations in metabolic inflammatory diseases. FEBS Lett. 2017;591(19):2959–77. https://doi.org/10.1002/1873-3468.12850.
4. Peng YC, Kuo F, Breiding DE, Wang YF, Mansur CP, Androphy EJ. AMF1 (GPS2) modulates p53 transactivation. Mol Cell Biol. 2001;21(17):5913–24. https://doi.org/10.1128/mcb.21.17.5913-5924.2001.
5. Zhang D, Harry GJ, Blackshear PJ, Zeldin DC. G-protein pathway suppressor 2 (GPS2) interacts with the regulatory factor X4 variant 3 (RFX4_v3) and functions as a transcriptional co-activator. J Biol Chem. 2008;283(13):8580–90. https://doi.org/10.1074/jbc.M708209200.
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