Phase II open label trial of imatinib in polycythemia rubra vera
Author:
Publisher
Springer Science and Business Media LLC
Subject
Hematology
Link
http://link.springer.com/content/pdf/10.1007/s12185-008-0193-1.pdf
Reference25 articles.
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2. Dai CH, Krantz SB, Koury ST, et al. Polycythemia vera. IV. Specific binding of stem cell factor to normal and polycythemia vera highly purified erythroid progenitor cells. Br J Haematol. 1994;88:497–505. doi: 10.1111/j.1365-2141.1994.tb05065.x .
3. Dai CH, Krantz SB, Sawyer ST. Polycythemia vera. V. Enhanced proliferation and phosphorylation due to vanadate are diminished in polycythemia vera erythroid progenitor cells; a possible defect of phosphatase activity in polycythemia vera. Blood. 1997;89:3574–81.
4. James C, Ugo V, Le Couedic JP, Staerk J, Delhommeau F, Lacout C, et al. A unique clonal JAK2–V617F2 mutation leading to constitutive signaling causes polycythemia vera. Nature. 2005;434:1144–8. doi: 10.1038/nature03546 .
5. Baxter EJ, Scott LM, Campbell PJ, East C, Fourouclas N, Swanton S, et al. Cancer Genome Project. Acquired mutation of the tyrosine kinase JAK2–V617F2 in human myeloproliferative disorders. Lancet. 2005;365:1054–61.
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