Identification of unbalanced genome copy number abnormalities in patients with multiple myeloma by single-nucleotide polymorphism genotyping microarray analysis-Reference-Cited by-同舟云学术

Identification of unbalanced genome copy number abnormalities in patients with multiple myeloma by single-nucleotide polymorphism genotyping microarray analysis

Published:2012-09-13 Issue:4 Volume:96 Page:492-500
ISSN:0925-5710
Container-title:International Journal of Hematology
language:en
Short-container-title:Int J Hematol

Author:

Kamada Yuhei,Sakata-Yanagimoto Mamiko,Sanada Masashi,Sato-Otsubo Aiko,Enami Terukazu,Suzukawa Kazumi,Kurita Naoki,Nishikii Hidekazu,Yokoyama Yasuhisa,Okoshi Yasushi,Hasegawa Yuichi,Ogawa Seishi,Chiba Shigeru

Publisher

Springer Science and Business Media LLC

Subject

Hematology

Link

http://link.springer.com/content/pdf/10.1007/s12185-012-1171-1.pdf

Reference50 articles.

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2. Chng WJ, et al. Genetic events in the pathogenesis of multiple myeloma. Best Pract Res Clin Haematol. 2007;20(4):571–96.

3. Largo C, et al. Multiple myeloma primary cells show a highly rearranged unbalanced genome with amplifications and homozygous deletions irrespective of the presence of immunoglobulin-related chromosome translocations. Haematologica. 2007;92(6):795–802.

4. Flactif M, et al. Interphase fluorescence in situ hybridization (FISH) as a powerful tool for the detection of aneuploidy in multiple myeloma. Leukemia. 1995;9(12):2109–14.

5. Chang H, et al. Frequent monoallelic loss of D13S319 in multiple myeloma patients shown by interphase fluorescence in situ hybridization. Leukemia. 1999;13(1):105–9.

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