Three atypical BCR/ABL transcripts detected simultaneously in a Philadelphia-positive acute lymphoblastic leukemia patient showing resistance to tyrosine kinase inhibitors-Reference-Cited by-同舟云学术

Three atypical BCR/ABL transcripts detected simultaneously in a Philadelphia-positive acute lymphoblastic leukemia patient showing resistance to tyrosine kinase inhibitors

Published:2022-09-10 Issue:1 Volume:117 Page:134-136
ISSN:0925-5710
Container-title:International Journal of Hematology
language:en
Short-container-title:Int J Hematol

Author:

Sun Haimin,Yan Zeying,Zhang Sujiang^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Hematology

Link

https://link.springer.com/content/pdf/10.1007/s12185-022-03451-4.pdf

Reference9 articles.

1. Baccarani M, Rosti G, Soverini S. Chronic myeloid leukemia: the concepts of resistance and persistence and the relationship with the BCR-ABL1 transcript type. Leukemia. 2019;33:2358–64.

2. Chen L, Wu Y, You Y, Xiao M, Yao Y, Li W. A novel e8a2 BCR-ABL1 intronic fusion through insertion of a chromosome 22 BCR gene fragment into chromosome 9 in an atypical Philadelphia (Ph) chromosome chronic myeloid leukemia patient. Leuk Lymphoma. 2016;57:2930–3.

3. Chisti MM, Sanders DS. Chronic myeloid leukemia with b3a3 (e14a3) fusion: a rare BCR/ABL rearrangement presenting with thrombocytosis—does MTHFR polymorphism matter. Case Rep Oncol. 2018;11:485–92.

4. Duan MH, Li H, Cai H. A rare e13a3 (b2a3) BCR-ABL1 fusion transcript with normal karyotype in chronic myeloid leukemia: the challenges in diagnosis and monitoring minimal residual disease (MRD). Leuk Res. 2017;59:8–11.

5. Kearney L, Crampe M, Langabeer SE. Frequency and spectrum of atypical BCR-ABL1 transcripts in chronic myeloid leukemia. Exp Oncol. 2020;42:78–9.

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