Erweiterter RAS-Mutationsstatus
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Hematology
Link
http://link.springer.com/content/pdf/10.1007/s00761-014-2688-0.pdf
Reference10 articles.
1. Amado RG, Wolf M, Freeman D et al (2007) Wild-type KRAS is required for panitumumab efficacy in patients with metastatic colorectal cancer: results from a randomized, controlled trial. EJC Suppl 5:#7LB
2. Benvenuti S, Sartore-Bianchi A, Di Nicolantonio F et al (2007) Oncogenic activation of the RAS/RAF signalling pathway impairs the response of metastatic colorectal cancers to anti-epidermal growth factor receptor antibody therapies. Cancer Res 67:2643–2648
3. Dietel M, Tannapfel A, Baretton G et al (2008) Molekularpathologische Analyse des K-RAS-Mutationsstatus beim metastasierten kolorektalen Karzinom – Ein Beispiel prädiktiver Pathologie. Onkologe 14:405–408. doi:10.1007/s00761-008-1367-4
4. Douillard JY, Siena S, Cassidy J et al (2010) Randomized, phase III trial of panitumumab with infusional fluorouracil, leucovorin, and oxaliplatin (FOLFOX4) versus FOLFOX4 alone as first-line treatment in patients with previously untreated metastatic colorectal cancer: the PRIME study. J Clin Oncol 28:4697–4705
5. Douillard JY, Oliner KS, Siena S et al (2013) Panitumumab-FOLFOX4 treatment and RAS mutations in colorectal cancer. N Engl J Med 369:1023–1034
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