Implications of Transient Receptor Potential Cation Channels in Migraine Pathophysiology
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Physiology,General Neuroscience
Link
https://link.springer.com/content/pdf/10.1007/s12264-020-00569-5.pdf
Reference170 articles.
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2. Dodick DW. Migraine. Lancet 2018, 391: 1315–1330.
3. Ophoff RA, Terwindt GM, Vergouwe MN, van Eijk R, Oefner PJ, Hoffman SM, et al. Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4. Cell 1996, 87: 543–552.
4. Dichgans M, Freilinger T, Eckstein G, Babini E, Lorenz-Depiereux B, Biskup S, et al. Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine. Lancet 2005, 366: 371–377.
5. Julius D. TRP channels and pain. Annu Rev Cell Dev Biol 2013, 29: 355–384.
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