The role of GABAB mechanisms in animal models of absence seizures

Author:

Caddick Sarah J.,Hosford David A.

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Reference62 articles.

1. Avanzini G., Vergnes M., Spreafico R., and Marescaux C. (1993) Calcium dependent regulation of genetically determined spike and waves by the reticular thalamic nucleus of rats.Epilepsia 34, 1–7.

2. Avoli M. (1987) Mechanisms of generalized epilepsy with spike and wave discharge.EEG Suppl. 39, 184–190.

3. Avoli M. and Gloor P. (1982) Role of the thalamus in generalized penicillin epilepsy: observations on decorticated cats.Exp. Neurol. 77, 386–402.

4. Avoli M., Gloor P., Kostopoulos G., and Gotman J. (1983) An analysis of penicillin-induced generalized spike and wave discharges using simultaneous recordings of cortical and thalamic single neurons.J. Neurophysiol. 50, 819–837.

5. Banerjee P. K. and Snead O. C. III (1995) Presynaptic gamma-hydroxybutyric acid (GHB) and gamma-aminobutyric acid B (GABAB) receptor-mediated release of GABA and glutamate (GLU) in rat thalamic ventrobasal nucleus (VB): a possible mechanism for the generation of absence-like seizures induced by GHB.J. Pharmacol. Exp. Ther. 273(3), 1534–1543.

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