A patient with congenital nephrogenic diabetes insipidus due to AVPR2 mutation complicated by persisting polydipsia under hemodialysis treatment
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Earth and Planetary Sciences,General Environmental Science
Link
http://link.springer.com/content/pdf/10.1007/s13730-020-00549-8.pdf
Reference13 articles.
1. Bockenhauer D, Bichet DG. Nephrogenic diabetes insipidus. Curr Opin Pediatr. 2017;29(2):199–205.
2. Earley LE, Orloff J. The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus. J Clin Investig. 1962;41(11):1988–97.
3. Berl T, Raz A, Wald H, Horowitz J, Czaczkes W. Prostaglandin synthesis inhibition and the action of vasopressin: studies in man and rat. Am J Physiol. 1977;232(6):F529.
4. Stokes JB. Integrated actions of renal medullary prostaglandins in the control of water excretion. Am J Physiol. 1981;240(6):F471.
5. Ryu HH, Chung JH, Shin BC, Kim HL. Congenital nephrogenic diabetes insipidus with end-stage renal disease. Korean J Intern Med. 2015;30(2):259–61.
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1. Trichlormethiazide/vasopressin;Reactions Weekly;2021-06
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