Aspartate aminotransferase, malate dehydrogenase, and pyruvate carboxylase activities in rat cerebral synaptic and nonsynaptic mitochondria: Effects ofin vitro treatment with ammonia, hyperammonemia and hepatic encephalopathy

Author:

Faff-Michalak Lidia,Albrecht Jan

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Biochemistry

Reference59 articles.

1. Albrecht, J., and Hilgier, W. (1984). Brain carbonic anhydrase activity in rats in experimental hepatogenic encephalopathy.Neurosci. Lett. 45: 7?10.

2. Albrecht, J., Hilgier, W., Lazarewicz, J. W., Rafalowska, U., and Wysmyk-Cybula, U. (1988). Astrocytes in acute hepatic encephalopathy: Metabolic properties,and transport function. In Norenberg, M. D., Hertz, L., and Schousboe, A. (eds),The Biochemical Pathology of Astrocytes, Alan R. Liss, New York, pp. 465?476.

3. Baraldi, M., and Zeneroli, M. L. (1982). Experimental hepatic encephalopathy: Changes in the binding of g-aminobutyric acid in rats.Science Wash. DC 216: 427?429.

4. Benjamin, A. M. (1982). Ammonia. In Lajtha, A. (ed),Handbook of Neurochemistry, 2nd Ed., Plenum Press, New York, pp. 117?137.

5. Bradford, H. F., Ward, H. K., and Foley, P. (1989). Glutaminase inhibition and the release of neurotransmitter glutamate from synaptosomes.Brain Res. 476: 29?34.

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