Long-term observations in gerbil brain following transient cerebral ischemia: Autoradiographic and histological study
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/BF00996929.pdf
Reference32 articles.
1. Araki, T., Kato, H. and Kogure, K. (1989). Selective neuronal vulnerability following transient cerebral ischemia in the gerbils: distribution and time course.Acta. Neurol. Scand. 80:548?553.
2. Araki, T., Kato, H. and Kogure, K. (1990). Neuronal damage and calcium accumulation following repeated brief cerebral ischemia in the gerbil.Brain Res. 528:114?122.
3. Araki, T., Kato, H. and Kogure, K. (1991a). Alteration of second messenger systems after transient cerebral ischemia in gerbils: protective effect of pentobarbital and an autoradiographic analysis.Neurosci. Lett. 130:57?60.
4. Araki, T., Kato, H., Hara, H. and Kogure, K. (1991b). Postischemic alteration of [3H] forskolin binding sites in selectively vulnerable areas: an autoradiographic study of gerbil brain.Neurosci. Lett. 125:159?162.
5. Araki, T., Kato, H., Kogure, K. and Saito, T. (1991c). Postischemic alteration of muscarinic acetylcholine, adenosine A1 and calcium antagonist binding sites in selectively vulnerable areas: an autoradiographic study of gerbil brain.J. Neurol. Sci. 106:206?212.
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1. Alteration of second messengers during acute cerebral ischemia – adenylate cyclase, cyclic AMP-dependent protein kinase, and cyclic AMP response element binding protein;Progress in Neurobiology;2001-10
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