Erythrocyte sodium-lithium countertransport activity is related to membrane fluidity in IDDM patients
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/BF00408477.pdf
Reference34 articles.
1. Jensen JS, Mathiesen ER, Norgaard K et al. (1990) Increased blood pressure and erythrocyte sodium/lithium countertransport activity are not inherited in diabetic nephropathy. Diabetologia 33: 619?624
2. Elving LD, Wetzels JFM, deNobel E, Berden JHM (1991) Erythrocyte sodium-lithium countertransport is not different in type 1 (insulin-dependent) diabetic patients with and without nephropathy. Diabetologia 34: 126?128
3. Carr SJ, Mbanya J-C, Thomas TH et al. (1990) Increased glomerular filtration rate in patients with insulin-dependent diabetes and elevated erythrocyte sodium-lithium countertransport. N Engl J Med 322: 500?505
4. Rutherford PA, Thomas TH, Carr SJ, Taylor R, Wilkinson R (1992) Kinetics of sodium-lithium countertransport activity in patients with uncomplicated type 1 diabetes. Clin Sci 82: 291?299
5. Testa I, Rabini RA, Fumelli P, Bertoli E, Mazzanti L (1988) Abnormal membrane fluidity and acetylcholinesterase activity in erythrocytes from insulin-dependent diabetic patients. J Clin Endocrinol Metab 67: 1129?1133
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2. Sodium-lithium countertransport is increased in normoalbuminuric type 1 diabetes but is not related to other risk factors for microangiopathy;European Journal of Clinical Investigation;2002-02
3. Abnormal thiol group modulation of sodium‒ lithium countertransport and membrane fluidity is associated with a disturbed relationship between serum triacylglycerols and membrane function in Type II diabetes;Clinical Science;2000-06-01
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