Decreased cysteine uptake by EAAC1 gene deletion exacerbates neuronal oxidative stress and neuronal death after traumatic brain injury

Author:

Choi Bo Young,Kim In Yeol,Kim Jin Hee,Lee Bo Eun,Lee Song Hee,Kho A. Ra,Jung Hee Jae,Sohn Min,Song Hong Ki,Suh Sang Won

Funder

National Research Foundation of Korea (NRF) grant funded by the Korea government (MEST)

Hallym University Specialization Fund

Publisher

Springer Science and Business Media LLC

Subject

Organic Chemistry,Clinical Biochemistry,Biochemistry

Reference45 articles.

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2. Aoyama K, Watabe M, Nakaki T (2008) Regulation of neuronal glutathione synthesis. J Pharmacol Sci 108(3):227–238. pii: JST.JSTAGE/jphs/08R01CR

3. Bains JS, Shaw CA (1997) Neurodegenerative disorders in humans: the role of glutathione in oxidative stress-mediated neuronal death. Brain Res Brain Res Rev 25(3):335–358. pii: S0165017397000453

4. Banbury-Conference (1997) Mutant mice and neuroscience: recommendations concerning genetic background. Banbury Conference on genetic background in mice. Neuron 19(4):755–759. pii: S0896-6273(00)80958-7

5. Bullock R, Kuroda Y, Teasdale GM, McCulloch J (1992) Prevention of post-traumatic excitotoxic brain damage with NMDA antagonist drugs: a new strategy for the nineties. Acta Neurochir Suppl (Wien) 55:49–55

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