Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice-Reference-Cited by-同舟云学术

Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice

Published:2010-07-30 Issue:3 Volume:40 Page:819-828
ISSN:0939-4451
Container-title:Amino Acids
language:en
Short-container-title:Amino Acids

Author:

Marsh Susan A.,Dell’Italia Louis J.,Chatham John C.

Publisher

Springer Science and Business Media LLC

Subject

Organic Chemistry,Clinical Biochemistry,Biochemistry

Link

http://link.springer.com/content/pdf/10.1007/s00726-010-0699-8.pdf

Reference53 articles.

1. Boudina S, Abel ED (2007) Diabetic cardiomyopathy revisited. Circulation 115:3213–3223

2. Brownlee M (2001) Biochemistry and molecular cell biology of diabetic complications. Nature 414:813–820

3. Champattanachai V, Marchase RB, Chatham JC (2007) Glucosamine protects neonatal cardiomyocytes from ischemia–reperfusion injury via increased protein-associated O-GlcNAc. Am J Physiol Cell Physiol 292:C178–C187

4. Chatham JC, Marchase RB (2010) The role of protein O-linked beta-N-acetylglucosamine in mediating cardiac stress responses. Biochim Biophys Acta 1800:57–66

5. Chatham JC, Forder JR, McNeill JH (eds) (1996) The Heart in Diabetes. Kluwer Academic Publishers, Norwell

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