Metabolic control of the K+ channel of human red cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology,Biophysics
Link
http://link.springer.com/content/pdf/10.1007/BF01871668.pdf
Reference44 articles.
1. Alvarez, J., Camaleño, J.M., García-Sancho, J., Herreros, B. 1986. Modulation of the Ca2+-dependent K+ transport by modifications of the NAD+/NADH ratio in intact human red cells.Biochim. Biophys. Acta 856:408–411
2. Alvarez, J., García-Sancho, J., Herreros, B. 1988. All or none cell responses of Ca2+-dependent K+ channels elicited by calcium or lead in human red cells can be explained by heterogeneity of agonist distribution.J. Membrane Biol. 104:129–138
3. Butcher, R.W., Sutherland, E.W. 1962. Adenosine 3′, 5′-phosphate in biological materials. I. Purification and properties of cyclic 3′, 5′-nucleotide phosphodiesterase and use of this enzyme to characterize adenosine 3′, 5′-phosphate in human urine.J. Biol. Chem. 237:1244–1250
4. Cook, D.L., Hales, C.N. 1984. Intracellular ATP directly blocks K+ channels in pancreatic β-cells.Nature (London) 311:271–273
5. Ewald, D., Williams, A., Levitan, I.B. 1985. Modulation of single Ca2+-dependent K+ channel activity by protein phosphorylation.Nature (London) 315:503–506
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