The isoprenoid end product N6-isopentenyladenosine reduces inflammatory response through the inhibition of the NFκB and STAT3 pathways in cystic fibrosis cells
Author:
Funder
Associazione Italiana per la Ricerca sul Cancro
Fondazione Umberto Veronesi
Università degli Studi di Salerno
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
http://link.springer.com/article/10.1007/s00011-017-1123-6/fulltext.html
Reference49 articles.
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3. Bifulco M, Malfitano AM, Proto MC, Santoro A, Caruso MG, Laezza C. Biological and pharmacological roles of N6-isopentenyladenosine: an emerging anticancer drug. Anticancer Agents Med Chem. 2008;8:200–4.
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5. Fradejas N, Carlson BA, Rijntjes E, Becker NP, Tobe R, Schweizer U. Mammalian Trit1 is a tRNA([Ser]Sec)-isopentenyl transferase required for full selenoprotein expression. Biochem J. 2013;450:427–32.
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