Insights into the complex interactions between Rab22a and extracellular vesicles in cancers-Reference-Cited by-同舟云学术

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Insights into the complex interactions between Rab22a and extracellular vesicles in cancers

Published:2023-12-08 Issue:1 Volume:73 Page:99-110
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Huang Shenghao,Bao Yuxuan,Kong Lingjie,Gao Sheng,Hua Chunyan

Funder

Science and Technology Plan Project of Wenzhou, China

National Natural Science Foundation of China

Natural Science Foundation of Zhejiang Province

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology

Link

https://link.springer.com/content/pdf/10.1007/s00011-023-01821-0.pdf

Reference57 articles.

1. Hutagalung AH, Novick PJ. Role of Rab GTPases in membrane traffic and cell physiology. Physiol Rev. 2011;91(1):119–49.

2. Qi S, Su L, Li J, Zhao P, Zhang Q, Niu X, et al. YIPF2 is a novel Rab-GDF that enhances HCC malignant phenotypes by facilitating CD147 endocytic recycle. Cell Death Dis. 2019;10(6):462.

3. Gao Y, Zheng X, Chang B, Lin Y, Huang X, Wang W, et al. Intercellular transfer of activated STING triggered by RAB22A-mediated non-canonical autophagy promotes antitumor immunity. Cell Res. 2022;32(12):1086–104.

4. Wang T, Gilkes DM, Takano N, Xiang L, Luo W, Bishop CJ, et al. Hypoxia-inducible factors and RAB22A mediate formation of microvesicles that stimulate breast cancer invasion and metastasis. Proc Natl Acad Sci U S A. 2014;111(31):E3234–42.

5. Ao X, Zou L, Wu Y. Regulation of autophagy by the Rab GTPase network. Cell Death Differ. 2014;21(3):348–58.

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