Targeting JAK/STAT signaling pathways in treatment of inflammatory bowel disease
Author:
Funder
National Key Research and Development Project of China
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
https://link.springer.com/content/pdf/10.1007/s00011-021-01482-x.pdf
Reference92 articles.
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2. Roda G, Chien Ng S, Kotze PG, Argollo M, Panaccione R, Spinelli A, et al. Crohn’s disease. Nat Rev Dis Primers. 2020;6:22. https://doi.org/10.1038/s41572-020-0156-2.
3. Bernstein CN, Burchill C, Targownik LE, Singh H, Roos LL. Events within the first year of life, but not the neonatal period, affect risk for later development of inflammatory bowel diseases. Gastroenterology. 2019;156:2190–7. https://doi.org/10.1053/j.gastro.2019.02.004.
4. Hedin CRH, Vavricka SR, Stagg AJ, Schoepfer A, Raine T, Puig L, et al. The pathogenesis of extraintestinal manifestations: implications for IBD research, diagnosis, and therapy. J Crohn’s Colitis. 2019;13:541–54. https://doi.org/10.1093/ecco-jcc/jjy191.
5. Aschenbrenner D, Quaranta M, Banerjee S, Ilott N, Jansen J, Steere B, et al. Deconvolution of monocyte responses in inflammatory bowel disease reveals an IL-1 cytokine network that regulates IL-23 in genetic and acquired IL-10 resistance. Gut. 2020. https://doi.org/10.1136/gutjnl-2020-321731.
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