Severity of critical illness correlates with CD3-low expression on iNKT-cells among septic surgical patients
Author:
Funder
National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
https://link.springer.com/content/pdf/10.1007/s00011-023-01795-z.pdf
Reference7 articles.
1. Torina A, Guggino G, La Manna M, Sireci G. The Janus face of NKT cell function in autoimmunity and infectious diseases. Int J Mol Sci. 2018;19(2):E440.
2. Kulkarni R, Villanueva A, Read L, Brisbin J, Bhaumik S, LaMarre J, et al. CpG oligonucleotide-mediated co-simulation of mouse invariant Natural Killer T-cells negatively regulates their activation status. Cell Tissue Res. 2017;369(3):541–54.
3. Wilson M, Johansson C, Olivares-Villagomez D, Singh A, Stanic A, Wang C, et al. The response of natural killer T cells to glycolipid antigens is characterized by surface receptor down-modulation and expansion. Proc Natl Acad Sci. 2003;100(19):10913–8.
4. Anantha R, Mazzuca D, Xu S, Porcelli S, Fraser D, Martin C, et al. T helper type-2 polarized invariant natural killer T cells reduce disease severity in acute intra-abdominal sepsis. Clin Exp Immunol. 2014;178(2):292–309.
5. Matulis G, Sanderson J, Lissin N, Asparuhova M, Bommineni G, Schumperli D, et al. Innate-like control of human iNKT-cell autoreactivity via the hypervariable CDR3beta loop. PLoS Biol. 2010;8(6):e1000402.
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