NLRP3 inflammasome activation and interleukin-1β release in macrophages require calcium but are independent of calcium-activated NADPH oxidases
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
http://link.springer.com/content/pdf/10.1007/s00011-014-0756-y.pdf
Reference54 articles.
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2. Carta S, Tassi S, Pettinati I, Delfino L, Dinarello CA, Rubartelli A. The rate of interleukin-1beta secretion in different myeloid cells varies with the extent of redox response to Toll-like receptor triggering. J Biol Chem. 2011;286(31):27069–80. doi: 10.1074/jbc.M110.203398 .
3. Thastrup O, Dawson AP, Scharff O, Foder B, Cullen PJ, Drobak BK, et al. Thapsigargin, a novel molecular probe for studying intracellular calcium release and storage. Agents Actions. 1989;27(1–2):17–23.
4. Brayden DJ, Hanley MR, Thastrup O, Cuthbert AW. Thapsigargin, a new calcium-dependent epithelial anion secretagogue. Br J Pharmacol. 1989;98(3):809–16.
5. Martinon F, Burns K, Tschopp J. The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. Mol Cell. 2002;10(2):417–26.
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