TRIM25-mediated XRCC1 ubiquitination accelerates atherosclerosis by inducing macrophage M1 polarization and programmed death-Reference-Cited by-同舟云学术

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TRIM25-mediated XRCC1 ubiquitination accelerates atherosclerosis by inducing macrophage M1 polarization and programmed death

Published:2024-06-19 Issue:9 Volume:73 Page:1445-1458
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Wu Hongxian,Gao Wei,Ma Yuanji,Zhong Xin,Qian Juying,Huang Dong,Ge Junbo

Funder

Natural Science Foundation of Shanghai

National Natural Science Foundation

Shanghai Clinical Research Physician Training Program

Shanghai Clinical Technology Innovation Program

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s00011-024-01906-4.pdf

Reference36 articles.

1. Martin SS, Aday AW, Almarzooq ZI, Anderson CAM, Arora P, Avery CL, et al. 2024 Heart Disease and Stroke statistics: a report of US and Global Data from the American Heart Association. Circulation. 2024;149(8):e347–913.

2. Libby P. The changing landscape of atherosclerosis. Nature. 2021;592(7855):524–33.

3. Li H, Cao Z, Wang L, Liu C, Lin H, Tang Y, Yao P. Macrophage subsets and death are responsible for atherosclerotic plaque formation. Front Immunol. 2022;13:843712.

4. Hou P, Fang J, Liu Z, Shi Y, Agostini M, Bernassola F, Bove P, Candi E, Rovella V, Sica G, et al. Macrophage polarization and metabolism in atherosclerosis. Cell Death Dis. 2023;14(10):691.

5. Oumouna-Benachour K, Hans CP, Suzuki Y, Naura A, Datta R, Belmadani S, et al. Poly(ADP-ribose) polymerase inhibition reduces atherosclerotic plaque size and promotes factors of plaque stability in apolipoprotein E-deficient mice: effects on macrophage recruitment, nuclear factor-kappab nuclear translocation, and foam cell death. Circulation. 2007;115(18):2442–50.

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