Potential role of NADPH oxidase-mediated activation of Jak2/Stat3 and mitogen-activated protein kinases and expression of TGF-β1 in the pathophysiology of acute pancreatitis-Reference-Cited by-同舟云学术

Potential role of NADPH oxidase-mediated activation of Jak2/Stat3 and mitogen-activated protein kinases and expression of TGF-β1 in the pathophysiology of acute pancreatitis

Published:2011-04-21 Issue:8 Volume:60 Page:791-800
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Ju Kyung Don,Lim Joo Weon,Kim Kyung Hwan,Kim Hyeyoung

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology

Link

http://link.springer.com/content/pdf/10.1007/s00011-011-0335-4.pdf

Reference56 articles.

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2. Petrone WF, English DK, Wong K, McCord JM. Free radicals and inflammation: superoxide dependent activation of a neutrophil chemotactic factor in plasma. Proc Natl Acad Sci USA. 1980;77:1159–63.

3. Wisner J, Green DF, Renner I. Evidence for a role of oxygen derived free radicals in the pathogenesis of cerulein induced acute pancreatic in rat. Gut. 1988;29:1516–23.

4. Hofbauer B, Saluja AK, Lerch MM, Bhagat L, Bhatia M, Lee HS, Frossard JL, Adler G, Steer ML. Intra-acinar cell activation of trypsinogen during cerulein-induced pancreatitis in rat. Am J Physiol. 1998;275:G352–62.

5. Lerch MM, Adler G. Experimental animal model of acute pancreatitis. Int J Pancreatol. 1994;15:159–70.

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