Blockade of macrophage-associated programmed death 1 inhibits the pyroptosis signalling pathway in sepsis
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
https://link.springer.com/content/pdf/10.1007/s00011-021-01493-8.pdf
Reference31 articles.
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2. Vu CTB, Thammahong A, Leelahavanichkul A, Ritprajak P. Alteration of macrophage immune phenotype in a murine sepsis model is associated with susceptibility to secondary fungal infection. Asian Pac J Allergy Immunol. 2019 https://doi.org/10.12932/AP-170519-0565
3. Xiao F, Wang D, Kong L, Li M, Feng Z, Shuai B, et al. Intermedin protects against sepsis by concurrently re-establishing the endothelial barrier and alleviating inflammatory responses. Nat Commun. 2018;9(1):2644.
4. Fallon EA, Chun TT, Young WA, Gray C, Ayala A, Heffernan DS. Program cell death receptor-1-mediated invariant natural killer T-cell control of peritoneal macrophage modulates survival in neonatal sepsis. Front Immunol. 2017;8:1469.
5. Gianchecchi E, Delfino DV, Fierabracci A. Recent insights into the role of the PD-1/PD-L1 pathway in immunological tolerance and autoimmunity. Autoimmun Rev. 2013;12(11):1091–100.
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