Potential role for ET-2 acting through ETA receptors in experimental colitis in mice

Author:

Claudino R. F.,Leite D. F.,Bento A. F.,Chichorro J. G.,Calixto J. B.,Rae G. A.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology

Reference70 articles.

1. Wallace KL, Zheng LB, Kanazawa Y, Shih DQ. Immunopathology of inflammatory bowel disease. World J Gastroenterol. 2014;20:6–21.

2. Sartor RB. Mechanisms of disease: pathogenesis of Crohn’s disease and ulcerative colitis. Nat Clin Pract Gastroenterol Hepatol. 2006;3:390–407.

3. Masaki T. Historical review: endothelin. Trends Pharmacol Sci. 2004;25:219–24.

4. Letizia C, Boirivant M, De Toma G, Cerci S, Subioli S, Scuro L, et al. Plasma levels of endothelin-1 in patients with Crohn’s disease and ulcerative colitis. Ital J Gastroenterol Hepatol. 1998;30:266–9.

5. Kanazawa S, Tsunoda T, Onuma E, Majima T, Kagiyama M, Kikuchi K. VEGF, basic-FGF, and TGF-beta in Crohn’s disease and ulcerative colitis: a novel mechanism of chronic intestinal inflammation. Am J Gastroenterol. 2001;96:822–8.

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