The macrophage senescence hypothesis: the role of poor heat shock response in pulmonary inflammation and endothelial dysfunction following chronic exposure to air pollution-Reference-Cited by-同舟云学术

The macrophage senescence hypothesis: the role of poor heat shock response in pulmonary inflammation and endothelial dysfunction following chronic exposure to air pollution

Published:2022-10-20 Issue:12 Volume:71 Page:1433-1448
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Costa-Beber Lílian Corrêa^ORCID,Guma Fátima Theresinha Costa Rodrigues

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology

Link

https://link.springer.com/content/pdf/10.1007/s00011-022-01647-2.pdf

Reference179 articles.

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2. Mehta S, et al. Ambient particulate air pollution and acute lower respiratory infections: a systematic review and implications for estimating the global burden of disease. Air Qual Atmos Health. 2013;6(1):69–83.

3. Furuyama A, et al. Extrapulmonary translocation of intratracheally instilled fine and ultrafine particles via direct and alveolar macrophage-associated routes. Arch Toxicol. 2009;83(5):429–37.

4. Hartiala J, et al. Ambient air pollution is associated with the severity of coronary atherosclerosis and incident myocardial infarction in patients undergoing elective cardiac evaluation. J Am Heart Assoc. 2016. https://doi.org/10.1161/JAHA.116.003947.

5. Xiong Q, et al. Mitochondrial dysfunction and inflammatory response in the cytotoxicity of NR8383 macrophages induced by fine particulate matter. Environ Toxicol Pharmacol. 2017;55:1–7.

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