IL-22 and IL-20 are key mediators of the epidermal alterations in psoriasis while IL-17 and IFN-γ are not-Reference-Cited by-同舟云学术

IL-22 and IL-20 are key mediators of the epidermal alterations in psoriasis while IL-17 and IFN-γ are not

Published:2009-03-30 Issue:5 Volume:87 Page:523-536
ISSN:0946-2716
Container-title:Journal of Molecular Medicine
language:en
Short-container-title:J Mol Med

Author:

Wolk Kerstin,Haugen Harald S.,Xu Wenfeng,Witte Ellen,Waggie Kim,Anderson Monica,vom Baur Elmar,Witte Katrin,Warszawska Katarzyna,Philipp Sandra,Johnson-Leger Caroline,Volk Hans-Dieter,Sterry Wolfram,Sabat Robert

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Drug Discovery,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00109-009-0457-0.pdf

Reference44 articles.

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2. Schon MP, Boehncke WH (2005) Psoriasis. N Engl J Med 352:1899–1912

3. Kupper TS, Fuhlbrigge RC (2004) Immune surveillance in the skin: mechanisms and clinical consequences. Nat Rev Immunol 4:211–222

4. Lew W, Bowcock AM, Krueger JG (2004) Psoriasis vulgaris: cutaneous lymphoid tissue supports T-cell activation and “Type 1” inflammatory gene expression. Trends Immunol 25:295–305

5. Wolk K, Sabat R (2006) Interleukin-22: a novel T- and NK-cell derived cytokine that regulates the biology of tissue cells. Cytokine Growth Factor Rev 17:367–380

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