Lovastatin induces apoptosis of k-ras-transformed thyroid cells via inhibition of ras farnesylation and by modulating redox state-Reference-Cited by-同舟云学术

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Lovastatin induces apoptosis of k-ras-transformed thyroid cells via inhibition of ras farnesylation and by modulating redox state

Published:2008-09-09 Issue:12 Volume:86 Page:1341-1351
ISSN:0946-2716
Container-title:Journal of Molecular Medicine
language:en
Short-container-title:J Mol Med

Author:

Laezza Chiara,Fiorentino Laura,Pisanti Simona,Gazzerro Patrizia,Caraglia Michele,Portella Giuseppe,Vitale Mario,Bifulco Maurizio

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Drug Discovery,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00109-008-0396-1.pdf

Reference38 articles.

1. Downward J (2003) Targeting RAS signalling pathways in cancer therapy. Nat Rev Cancer 3:11–22

2. Wittinghofer A, Nassar N (1996) How Ras-related proteins talk to their effectors. Trends Biochem Sci 21:488–491

3. Rassool FV, Gaymes TJ, Omidvar N, Brady N, Beurlet S, Pla M, Reboul M, Lea N, Chomienne C, Thomas NS, Mufti GJ, Padua RA (2007) Reactive oxygen species, DNA damage, and error-prone repair: a model for genomic instability with progression in myeloid leukemia? Cancer Res 67(18):8762–8771

4. Santillo M, Mondola P, Serù R, Annella T, Cassano S, Ciullo I, Tecce MF, Iacomino G, Damiano S, Cuda G (2001) Opposing functions of Ki- and Ha-Ras genes in the regulation of redox signals. Current Biology 11:614–619

5. Casey PJ, Solski PA, Der CJ, Buss JE (1989) p21ras is modified by a farnesyl isoprenoid. Proc Natl Acad Sci USA 86:8323–8327

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