The NAv1.7 blocker protoxin II reduces burn injury-induced spinal nociceptive processing-Reference-Cited by-同舟云学术

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The NAv1.7 blocker protoxin II reduces burn injury-induced spinal nociceptive processing

Published:2017-10-23 Issue:1 Volume:96 Page:75-84
ISSN:0946-2716
Container-title:Journal of Molecular Medicine
language:en
Short-container-title:J Mol Med

Author:

Torres-Pérez Jose Vicente,Adamek Pavel,Palecek Jiri,Vizcaychipi Marcela,Nagy Istvan^ORCID,Varga Angelika

Funder

Chelsea and Westminster Health Charity

Grantová Agentura České Republiky

Ministerstvo Školství, Mládeže a Tělovýchovy

Grantová Agentura, Univerzita Karlova

Akademie Věd České Republiky

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Drug Discovery,Molecular Medicine

Link

http://link.springer.com/article/10.1007/s00109-017-1599-0/fulltext.html

Reference33 articles.

1. Laycock H, Valente J, Bantel C, Nagy I (2013) Peripheral mechanisms of burn injury-associated pain. Eur J Pharmacol 716:169–178

2. Julius D, Basbaum AI (2001) Molecular mechanisms of nociception. Nature 413:203–210

3. Eijkelkamp N, Linley JE, Baker MD, Minett MS, Cregg R, Werdehausen R, Rugiero F, Wood JN (2012) Neurological perspectives on voltage-gated sodium channels. Brain 135:2585–2612

4. Shields SD, Cheng X, Uceyler N, Sommer C, Dib-Hajj SD, Waxman SG (2012) Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury. J Neurosci 32:10819–10832

5. Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, Wood JN (2004) Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain. Proc Natl Acad Sci U S A 101:12706–12711

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5. Fluorescent‐ and tagged‐protoxin II peptides: potent markers of the Na v 1.7 channel pain target;British Journal of Pharmacology;2021-05-14

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