Hearts of this ILK rely on TNNI3K, a MAPKKK that regulates TNNI3
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics (clinical),Drug Discovery,Molecular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00109-003-0436-9.pdf
Reference2 articles.
1. Zhao Y, Meng X-M, Wei Y-J, Zhao X-W, Liu D-Q, Cao H-Q, Liew C-C, Ding J-F (2003) Cloning and characterization of a novel cardiac-specific kinase that interacts specifically with cardiac troponin I. J Mol Med (http://dx.doi.org/10.1007/s00109-003-0427-x)
2. Seidman JG, Seidman C (2001) The genetic basis for cardiomyopathy from mutation identification to mechanistic paradigms. Cell 104:557–567
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1. The Diverse Roles of TNNI3K in Cardiac Disease and Potential for Treatment;International Journal of Molecular Sciences;2021-06-15
2. 3D-QSAR Assisted Design of Novel 7-Deazapurine Derivatives as TNNI3K Kinase Inhibitors Using Molecular Docking and Molecular Dynamics Simulation;Letters in Drug Design & Discovery;2020-02-13
3. Overexpression of Cardiac-Specific Kinase TNNI3K Promotes Mouse Embryonic Stem Cells Differentiation into Cardiomyocytes;Cellular Physiology and Biochemistry;2017
4. Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction;Journal of Molecular and Cellular Cardiology;2013-01
5. Evidence, hypotheses and significance of MAP kinase TNNI3K interacting with its partners;World Journal of Hypertension;2012
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