Sacubitril/valsartan reduces endoplasmic reticulum stress in a rat model of doxorubicin-induced cardiotoxicity
Author:
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00204-022-03241-1.pdf
Reference33 articles.
1. Bhattarai KR, Chaudhary M, Kim H-R, Chae H-J (2020) Endoplasmic reticulum (ER) stress response failure in diseases. Trends Cell Biol 30(9):672–675. https://doi.org/10.1016/j.tcb.2020.05.004
2. Bosch X, Rovira M, Sitges M et al (2013) Enalapril and carvedilol for preventing chemotherapy-induced left ventricular systolic dysfunction in patients with malignant hemopathies: the OVERCOME trial (preventiOn of left Ventricular dysfunction with Enalapril and caRvedilol in patients submitted to intensive ChemOtherapy for the treatment of Malignant hEmopathies). J Am Coll Cardiol 61(23):2355–2362. https://doi.org/10.1016/j.jacc.2013.02.072
3. Boutagy NE, Feher A, Pfau D et al (2020) Dual angiotensin receptor-neprilysin inhibition with sacubitril/valsartan attenuates systolic dysfunction in experimental doxorubicin-induced cardiotoxicity. JACC CardioOncol 2(5):774–787. https://doi.org/10.1016/j.jaccao.2020.09.007
4. Cardinale D, Colombo A, Bacchiani G et al (2015) Early detection of anthracycline cardiotoxicity and improvement with heart failure therapy. Circulation 131(22):1981–1988. https://doi.org/10.1161/circulationaha.114.013777
5. Chang P, Zhang M, Zhang X et al (2019) B-type natriuretic peptide attenuates endoplasmic reticulum stress in H9c2 cardiomyocytes underwent hypoxia/reoxygenation injury under high glucose/high fat conditions. Peptides 111:103–111. https://doi.org/10.1016/j.peptides.2018.04.016
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