Dietary fructose as a risk factor for non-alcoholic fatty liver disease (NAFLD)-Reference-Cited by-同舟云学术

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Dietary fructose as a risk factor for non-alcoholic fatty liver disease (NAFLD)

Published:2016-12-19 Issue:4 Volume:91 Page:1545-1563
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Alwahsh Salamah Mohammad,Gebhardt Rolf

Funder

The Bundesministerium für Forschung und Technologie (BMBF) in the framework of the Systems Biology initiative “Virtual Liver Network”

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00204-016-1892-7.pdf

Reference166 articles.

1. Abdelmalek MF, Suzuki A, Guy C et al (2010) Increased fructose consumption is associated with fibrosis severity in patients with nonalcoholic fatty liver disease. Hepatology 51:1961–1971. doi: 10.1002/hep.23535

2. Abdelmalek MF, Lazo M, Horska A et al (2012) Higher dietary fructose is associated with impaired hepatic adenosine triphosphate homeostasis in obese individuals with type 2 diabetes. Hepatology 56:952–960. doi: 10.1002/hep.25741

3. Agrawal R, Noble E, Vergnes L et al (2015) Dietary fructose aggravates the pathobiology of traumatic brain injury by influencing energy homeostasis and plasticity. J Cereb Blood Flow Metab 36:941–953. doi: 10.1177/0271678X15606719

4. Ahmad S, Sultan S, Naz N et al (2014) Regulation of iron uptake in primary culture rat hepatocytes: the role of acute-phase cytokines. Shock 41:337–345. doi: 10.1097/SHK.0000000000000107

5. Albuquerque D, Estévez MN, Víbora PB et al (2014) Novel variants in the MC4R and LEPR genes among severely obese children from the iberian population. Ann Hum Genet 78:195–207. doi: 10.1111/ahg.12058

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